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Cerebral amyloid angiopathy (CAA), characterized by deposition of amyloid in the small arteries and arterioles of the cerebral cortex and leptomeninges, leads to superficial bleeding in lobar brain regions or the subarachnoid space. There are no disease-modifying treatments, and it has the highest recurrence rate of any stroke syndrome, with an average annual 7.4% risk of recurrent intracerebral hemorrhage (ICH) after an initial ICH.1
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