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Left ventricular wall motion abnormalities (LVWMAs) are commonly observed in a variety of medical conditions, including coronary artery disease, congestive heart failure, stress-induced cardiomyopathy, myocarditis, chronic renal disease, and stroke.1–4 Their underlying disease mechanisms and their potential causative role for stroke remain inadequately elucidated. LVWMAs may directly increase stroke risk through thrombus formation in the left ventricle5 or may merely indicate the burden of systemic atherosclerosis underlying stroke, and not the primum movens.
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