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Posterior cortical atrophy (PCA) is a dementing syndrome that presents with signs and symptoms of cortical visual dysfunction.1 The clinical features of PCA reflect dysfunction of both the ventral/occipito-temporal visual processing pathway causing apperceptive visual agnosia, alexia, and prosopagnosia and dorsal/occipito-parietal pathway causing Balint syndrome (simultanagnosia, optic ataxia and ocular apraxia), transcortical sensory aphasia, apraxia, and some or all of the elements of Gerstmann syndrome (agraphia, acalculia, finger agnosia, right-left disorientation).2,3 Frontal lobe functions and memory are relatively preserved until later in the course of the disease.
In this issue of Neurology, McMonagle et al.4 describe the cognitive profile of 19 patients with PCA and compare this to the profile of 11 patients with typical amnestic Alzheimer disease …
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