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To the Editor:
We read with interest the report on the suppression of myoclonus by levetiracetam (LEV) in two patients with post-anoxic myoclonus and one patient with post-encephalitic myoclonus.1 It confirms our previous findings in three patients with severe action myoclonus, including two with Unverricht–Lundborg disease (ULD) and one with post-anoxic myoclonus.2 The latter had the most spectacular effect, a sort of “Lazarus syndrome”: Within 24 hours, he was able to leave his wheelchair and could walk around freely. We have now used LEV in 10 patients with ULD, with very positive results. However, these results depend on earlier treatment with piracetam (PIR), a very potent antimyoclonic agent, the efficacy of which was reported many years ago, first in patients with the Lance–Adams …
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