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Paroxysmal kinesigenic choreoathetosis (PKC) is a clinical entity whose etiology and susceptibility to treatment with anticonvulsants remains poorly understood. Kertesz [1] introduced the term PKC to describe individuals with repeated brief paroxysms of dystonic, tonic, or choreoathetotic movements that were precipitated by sudden movements or by stressful or exciting states. These patients differ from those with symptomatic paroxysmal choreoathetosis, paroxysmal dystonia, paroxysmal dystonia of pregnancy, and familial frontal epilepsy. Attacks of PKC respond to antiepileptic agents. We report seven patients with familial or sporadic PKC who were exquisitely sensitive to very low doses and levels of carbamazapine Table 1. These doses and levels were much lower than what is required for seizure control in most individuals.
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Table 1. Clinical findings in seven patients with paroxysmal kinesigenic choreoathetosis
Case report.
At age 10, this 16-year-old right-handed girl began experiencing episodes of an "unwell feeling" followed by largeamplitude writhing movements of the left …
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