@article {Raposo697作者={尼古拉斯Raposo和M {\ ' e}拉妮Planton和帕特里斯P {\ ' e}跑和皮埃尔Payoux Fabrice博纳维尔和艾莎Lyoubi让弗兰{\ c c} ois Albucher和Blandine Acket和安妮苏菲Salabert Jean Marc Olivot和安妮Hitzel弗兰{\ c c} ois Chollet和J {\ ' r e} {\ ' e}米氏Pariente}, title = {Florbetapir成像在脑淀粉样angiopathy-related出血},体积={89}={7},页面= {697 - 704}= {2017},doi = {10.1212 / WNL。出版商0000000000004228}= {Wolters Kluwer健康,公司代表美国神经病学学会},文摘={目的:评估是否18 f-florbetapir,宠物淀粉指示剂,可以结合血管淀首页粉样蛋白在脑淀粉样血管病(CAA)通过比较皮质florbetapir保留患者的急性期期间CAA-related大叶性脑出血患者(我)和hypertension-related深的我。方法:急性CAA-related患者患者大叶性我是前瞻性的,相比之下,深的我。18 f-florbetapir宠物,大脑核磁共振,所有参与者均获得APOE基因型。皮质florbetapir标准摄入值比率(SUVr)计算整个小脑作为参考。CAA患者和那些与我比较深的意思是皮质florbetapir SUVr值。结果:15例急性肺叶的波士顿我完成修改后的标准可能的创新艺人经纪公司(平均年龄12年= 67 {\ textpm})和18例急性深我(平均年龄= 63 {\ textpm}) 11年了。全球平均皮质florbetapir SUVr明显高于患者CAA-related我比患者深我(0.12 vs 1.12 1.27 {\ textpm} {\ textpm} 0.12, p = 0.001)。皮质florbetapir SUVr分化CAA-ICH患者从那些深我(曲线下的面积= 0.811;95 \ %可信区间[CI] 0.642 {\ textendash} 0.980)的敏感性为0.733 (95 \ % CI 0.475 {\ textendash} 0.893)和特异性为0.833 (95 \ % CI 0.598 {\ textendash} 0.948)。结论:皮质florbetapir吸收增加患者CAA-related我相对于那些深的我。虽然18 f-florbetapir宠物可以标签血管β-amyloid和作为一个结果标志在以后的临床试验,其诊断价值在急性CAA-related我似乎在临床practice.Aβ=β-amyloid有限;广告=阿尔茨海默病; CAA=cerebral amyloid angiopathy; CI=confidence interval; CMB=cerebral microbleed; cSS=cortical superficial siderosis; FLAIR=fluid-attenuated inversion recovery; GRE=gradient recalled echo; ICH=intracerebral hemorrhage; IQCODE=Informant Questionnaire on Cognitive Decline in the Elderly; PiB=Pittsburgh compound B; ROI=region of interest; STRIVE=Standards for Reporting Vascular Changes on Neuroimaging; SUV=standardized uptake value; SUVr=standardized uptake value ratio; WMH=white matter hyperintensities}, issn = {0028-3878}, URL = {//www.ez-admanager.com/content/89/7/697}, eprint = {//www.ez-admanager.com/content/89/7/697.full.pdf}, journal = {Neurology} }