TY - T1的感染、炎症和脑血管缺血JF -神经学乔-神经病学SP - S47 LP - S51做- 10.1212 / WNL.49.5_Sup首页pl_4。S47六世- 49 - 5 5 4盟阿明j·格劳Y1 - 1997/11/01 UR - //www.ez-admanager.com/content/首页49/5_Suppl_4/S47.abstract N2 -止血和炎症通路相互相互作用。增加炎症参数的续集和脑血栓形成和缺血的危险因素。缺血性中风急性期反应,特点是瞬态纤维蛋白原升高,白细胞计数,和一些细胞因子。本文主要关注炎性变化先于脑缺血和炎症/感染危险因素中风和短暂性脑缺血发作(TIA)。炎症参数和血管性疾病和危险因素。白细胞计数是积极与缺血性中风和心肌梗死的风险。1、2粒细胞计数似乎主要贡献这样的风险升高。最近的流行病学研究发现,单核细胞计数也与心肌缺血的风险增加。3高血浆纤维蛋白原水平中风和心肌梗死的危险因素(MI)。4此外,几个血清糖蛋白,主要是急性期反应物被证明是与冠状动脉粥样硬化的严重程度相关。5虽然上述炎症参数似乎独立预测缺血性疾病的风险,一些血管危险因素本身与增加炎症相关参数。 We studied the association of leukocyte count, fibrinogen, and C-reactive protein (CRP) with vascular risk factors in the control group of our recent case-control study investigating acute infection as a risk factor for stroke.6,7 These control subjects were randomly selected from the population and were matched to stroke patients for sex and age. Subjects with the following conditions were excluded from the analysis: malignancies, acute or chronic inflammatory diseases; recent trauma, surgery, and vascular diseases. In an ANOVA model with several risk factors and ischemic diseases, diabetes mellitus was associated with higher leukocyte count, fibrinogen and CRP. High age was correlated with increased leukocyte count and fibrinogen; Smoking and a history of stroke … ER -